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Although those are the best for muscle growth, you will also see good development of muscles using S4 Andarine and LGD-4033 Ligandrolactone.
In fact, the research has demonstrated that using LIGAndarine for muscle enhancement does so to some extent.
Ligandrolactone has been used historically as an antifibrinolytic, and it has also been used for growth stimulation at high doses (1mg/kg) due to strong anti-oxidant effect. It is still used today for that purpose.
As with any stimulant, there is a higher level of risk with LIGAndarine.
LIGAndarine is a natural compound that may induce liver enzymes and have a synergistic effect with S4 and LGD-4033:
One study showed that adding LIGAndarine to human muscle injections induced a significant increase from 0.4% to 1.2% of ATP in the myotubes and that an increase in skeletal muscle cell mass could have been caused by LIGAndarine.
Some caution is advised when using either LIGAndarine or S4/LGD-4033; these products are often associated with hepatotoxicity due to their high doses, their low rate of absorption as compared to L-Glutamate, and their interactions with CYP3A4.
However, this may not apply to the higher doses and the low rate of absorption as a result of its high molecular weight (13-28nmol/L) and low pH. Inhibition of glycogen synthesis has also been observed after oral administration of LIGAndarine (1mg/kg) in rats.
In regards to liver enzymes, a mouse study in which 10 mg/kg of LIGAndarine were administered to Livers (Liver Liver) failed to show significant inhibition, and this was also replicated in an in vivo model using rat liver. While these studies do not show dose-dependent inhibition, when the LIGAndarine-Liver experiment was repeated (10mg/kg liver liver injections twice weekly for 3 weeks), it was found that the mice developed increased rates of liver damage compared to control. These mechanisms may be related to its potential hepatotoxicity in higher doses of the extract.
Although LIGAndarine is known to be bioactive, the mechanisms behind this are unknown. Further, the liver is notoriously poor in cholesterol metabolism when compared to other organs, which may be due to the lack of availability of L-Enzymatic
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